The next logical step in the treatment of Alzheimer’s
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University of British Columbia scientists may have uncovered a new explanation for how Alzheimer’s disease destroys the brain – a profusion of blood vessels.
While the death of cells, whether they are in the walls of blood vessels or in brain tissue, has been a major focus of Alzheimer’s disease research, a team led by Wilfred Jefferies, a professor in UBC’s Michael Smith Laboratories, has shown that the neurodegenerative disease might in fact be caused by the propagation of cells in blood vessel walls.
Examining brain tissue from mouse models of Alzheimer’s disease, Jefferies’ team found nearly double the density of capillaries compared to normal mice. They also found a similarly higher density of capillaries in brain samples of people who had died of the disease, compared to samples from people who didn’t have it.
Jefferies, in an article published online today by PLoS One, theorizes that the profusion of blood vessels is stimulated by amyloid beta, a protein fragment that has become a hallmark of Alzheimer’s disease. The blood vessel growth, or “neo-angiogenesis,” leads to a breakdown of the blood-brain barrier – the tightly interlocked network of cells that allows oxygen-carrying blood to reach brain tissue while blocking harmful substances, such as viruses. Continue to read: dailymail.co.uk
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