Sleep deprivation and increased stress may affect Alzheimer’s risk
(brain cell activity)
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Higher levels of cell chatter boost amyloid beta in the brain regions that Alzheimer’s hits first, researchers at Washington University School of Medicine in St. Louis report. Amyloid beta is the main ingredient of the plaque lesions that are a hallmark of Alzheimer’s.
These brain regions belong to a network that is more active when the brain is at rest. The discovery that cells in these regions communicate with each other more often than cells in other parts of the brain may help explain why these areas are frequently among the first to develop plaques, according to the researchers.
Working with mice genetically engineered to develop Alzheimer’s type-brain changes, scientists reduced the size and number of plaques by decreasing brain cell activity in certain regions.
The results, appearing May 1 in Nature Neuroscience, are the latest to hint at a resolution to lines of evidence that have suggested busier brain cells can both contribute to and prevent Alzheimer’s. According to a new theory, which brain cells are kept busy may make all the difference. Read more: wustl.edu
Not all beta-glucans are equal for immune health
The ability of beta-glucan to modulate immune function is related to the specific form of the beta-glucan, with not all beta-glucans offering the same benefits, says a new study in Nature. Read more: nutraingredients.com
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